The Cause of Alzheimer's Disease

Alzheimer's patients typically live from vanadium to 10 years after diagnosis, though with recent improvements in health cathexis, they can live for 15 years or more than. The unhealthiness is particularly stressful on those who grow to c be for these patients, particularly as the disease progresses. Risk factors for Alzheimer's include signal trauma, get on with, Down's syndrome, and a genetic mutation.

Familial Alzheimer's disease accounts for approximately 10 percent of cases, and virtually of these patients develop symptoms before age 40 (Gale, 2001). The early onset genes ar found on chromosomes 1, 14, and 21, and are termed presenilins. The gene on chromosome 14 appears to be primarily trusty for early onset Alzheimer's disease, which has a more rapid course. The presenilin on chromosome 1 seems to be responsible for Alzheimer's of later onset, with a more protracted course. Recently, genetic studies earn shown that the gene for apolipoprotein E (ApoE), a protein which moves cholesterol in the bloodstream and can bind to amyloid beta-protein, can affect the chances of getting Alzheimer's disease. There are threesome forms of ApoE - ApoE2, ApoE3, and ApoE4. They are normally occurring variants, and it has been found that the inheritance of ApoE4 increases the risk and lowers the age of onset of the disease, and inheritance of ApoE2 decreases the risk and raises the age of onset. The gene is

There usually comes a cadence when a family member cannot vex for an Alzheimer's disease patient at home, and the patient needs to be in long-term residential care (Growdon, Long-term, 2002). These additionalized nursing homes have facilities designed to modify life for Alzheimer's patients, such as low noise levels, compound lighting, plenty of walking space, outdoor courtyards to prevent patients wandering off, and color-coded hallways to dish up patients in finding their way. Many activities are provided to provide pith and enjoyment for patients.

There are choices of types of living facilities from nursing homes, to special care units within nursing homes, to assisted-living facilities, to retirement homes in which patients cam be moved to higher levels of service as their needs change. The macrocosm is that sooner or later, it will become impossible to care for an Alzheimer's disease patient at home.

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FDA advisory delegation recommends approval of Nameda. (2003, October 24). Drug Week, 23.

Antioxidants could reduce the damage to neurons from free radicals (Growdon, The Search, 2002). Excitotoxins are neurotransmitters that activate neurons, but in excessive amounts they may deflower them. Research has shown that excitotoxins and free radicals may be responsible for some of the damage to neurons in Alzheimer's disease and in Parkinson's disease. Studies have shown that selegiline (Eldepryl), an antioxidant, and vitamin E, a strong antioxidant, might modestly slow the development of Alzheimer's disease. Two recent studies showed that diets rich in vitamin E from food sources may help lower the risk of developing Alzheimer's disease.

Although no long-term, prospective studies have confirmed the data, some observational investigations have shown evidence that hormone replacement therapy slows down the onset of Alzheimer's disease in women (Growdon, The Search, 2002). Observational studies have al
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